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CLINICAL NEWS American College of Cardiology Extended Learning • Is the patient taking the prescribed medications? Poor treatment adherence, he said, accounts for about half of all failure in BP control. • Is the treatment failure real? He said at least one-third of “office resistant hypertension” is actually white-coat hypertension that can be confirmed by ambulatory BP monitoring.3 • Is an optimal treatment regimen being prescribed? Guidelines suggest that in the absence of a concomitant condition requiring particular drug classes or an established contraindication, patients with resistant hypertension should be on all three of these agents: 1) renin–angiotensin system blocker (angiotensin-converting enzyme inhibitor or angiotensin receptor blocker); 2) calcium channel blockers; and 3) thiazide diuretics. • Is the patient taking conflicting drugs? NSAIDs, cold remedies, weight loss medications, and antidepressants are among the drugs that can interfere with treatment and raise BP, sometimes sharply (TABLE).4 TABLE Medications that Can Interfere with Blood Pressure Control Non-steroidal anti-inflammatory drugs/COX-2 inhibitors Oral contraceptives (estrogen predominant) While catheter-based RDN failed spectacularly in a sham-controlled study, the approach may be down but not out. • 15 had secondary hypertension; • 17 were controlled on four drugs; • 40 were nonadherent (based on urinary analysis), with 30% taking none of their meds at all and the rest taking about half of what was prescribed. (At least there was consistency: lack of adherence was almost evenly distributed among different classes of antihypertensive drugs.); and Sympathomimetic agents (decongestants, diet pills, cocaine) Stimulants (amphetamines, methylphenidate) Alcohol Antidepressants (tricyclic antidepressants, serotonin and norepinephrine reuptake inhibitors) Cyclosporine Erythropoietin Natural licorice Herbal compounds (ephedra or ma huang) • Does the patient have a secondary form of hypertension (e.g., chronic kidney disease, sleep apnea, aldosterone excess)? Studies have suggested that the prevalence of idiopathic hyperaldosteronism ranges from 17% to 22% of individuals with resistant hypertension. • Could an additional drug help (spironolactone can be a good first step)? Other options: beta-blockers; centrally acting drugs like clonidine; alpha blockers; or direct vasodilators, such as hydralazine. To give you an idea of what you might expect from using these individual pieces of advice in a population of patients, Dr. Weber points to one study of 375 patients referred for specialist care due to uncontrolled hypertension despite being on three drugs.5 Of these: • 267 fell out of the study by maximizing doses and excluding white-coat hypertension; 30 CardioSource WorldNews • 36 were true resistant hypertensives (i.e., just 10% of the 375 referred patients). The investigators postulate that the shorter distance of nerve endings to the arterial lumen in the distal segment of the renal artery may account for the improved treatment efficacy. In To listen to an interan accompanying editorial comview with Michael A. Weber, MD, on ment,8 coauthored by Deepak the topic of unconBhatt, MD (first author the shamtrolled hypertencontrolled trial that brought an sion, scan the code. The interview was inglorious end to SYMPLICITY conducted by David HTN-39), “It appears that the adR. Holmes, MD. dition of distal lesion targets may be the most efficient approach for improving success in renal denervation, despite fewer nerves surrounding the distal vessel.” Bhatt and his coauthor Neal N. Sawlani, MD, added that ongoing studies in humans are currently underway using new approaches with multielectrode catheters, incorporating an ongoing accrual of knowledge (NCT02439775, NCT02439749, and NCT02392351). They concluded, “With new methodological standards and novel preclinical studies, catheter-based renal denervation is poised to undergo significant innovation in device application. Renal denervation appears to have reached a new branch point in its development.” Editor’s note: Dr. Weber recently published a paper on RDN for the treatment of hypertension titled, Making a new start, getting it right.10 REFERENCES: This is not an isolated study’s findings. In one recent trial evaluating patients before being sent for renal denervation (RDN), top tier French specialists searched through 1,416 referred resistant patients to find 106 eligible subjects who truly had resistant hypertension (7.5%).6 Once you subtract all those factors that get a patient classified as having resistant hypertension, Dr. Weber suspects the prevalence of resistant hypertension in those being treated for high blood pressure is, in reality, about 1%. “It’s a pretty rare diagnosis,” he said. REMEMBER RDN? While catheter-based RDN failed spectacularly in a sham-controlled study, the approach may be down but not out. Several reasons for the variability in response to renal artery denervation in humans have been posited, particularly limitations with the anatomic targets used for radiofrequency ablation in clinical trials. Recently in JACC, Mahfoud et al. reported that increasing the number of radiofrequency lesions in the main renal artery was not sufficient to yield a clear dose-response relationship. However, targeted treatment of the renal artery branches or distal segment of the main renal artery resulted in markedly less variability of response and significantly greater reduction of both norepinephrine and axon density than conventional treatment targeting only the main renal artery for RDN.7 1. Mozaffarian D, Benjamin EJ, Go AS, et al. Circulation. 2016;133:e38-e360. 2. Weber MA. Trends in Cardiovasc Med. 2015;25:755-6. 3. de la Sierra A, Segura J, Banegas JR, et al. Hypertension. 2011;57:898-902. 4. Calhoun DA, Jones D, Textor S, et al. Hypertension. 2008;51:1403-19. 5. Jung O, Gechter JL, Wunder C, et al. J Hypertens. 2013;31:766-74. 6. Azizi M, Sapoval M, Gosse P, et al. Lancet. 2015;385:1957-65. 7. Mahfoud F, Tunev S, Ewen S, et al. J Am Coll Cardiol. 2015;66:1766-75. 8. Sawlani NN, Bhatt DL. J Am Coll Cardiol. 2015;66:1776-1778. 9. Bhatt DL, Kandzari DE, O’Neill WW, et al. N Engl J Med. 2014;370:1393-401. 10. Weber MA, Kirtane A, Mauri L, Townsend RR, Kandzari DE, Leon MB. J Clin Hypertens (Greenwich). 2015;17:743-50. Take-aways • While more patients are qualifying as having “resistant hypertension,” only a small percentage of these patients will ultimately be determined to have truly resistant high blood pressure. • There are a number of discernible factors that can be detected and lead to effective treatment of what was originally thought to be “resistant hypertension.” • Renal denervation is down but not out. New studies are suggesting that this approach may still become an effective means of managing patients with truly resistant hypertension. May 2016