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Moonshot Progress SIDEBAR 2 ASH Task Force on Precision Medicine As part of its efforts to advance hematol- ogy research, the American Society of Hematology (ASH) established the Task Force on Precision Medicine to lead ef- forts in increasing the understanding of germline and somatic mutations in hema- tologic disorders. 1 A separate Task Force on Immunotherapy – a key focus of the Moonshot Initiative – was formed to ad- dress specific scientific and clinical issues related to this area of precision medicine. “When discussing germline mutations, the first challenge is knowing what genes have variants that lead to hematologic disease,” explained Task Force Co-Chair Benjamin L. Ebert, MD, PhD, chair of medical oncology at Dana-Farber Cancer Institute. “The second challenge is, once you sequence those genes, determining which variants are pathogenic and which don’t have an impact on the likelihood of developing disease.” The Task Force is approaching these challenges through the publication of a series of review articles designed to educate clinicians about the implications of sequencing studies and through the la- borious task of annotating all variants that clinicians might find in these hematologic disorders as benign or pathologic, Dr. Ebert explained. As part of that effort, ASH has partnered with the National Institutes of Health Clinical Genome Resource (ClinGen) to develop a broad and accessible compen- dium of genomic data aimed at improving the diagnosis of hematologic disorders. ASH is supporting two expert review panels to analyze the clinical significance of variants and mutations understood to be associated with myeloid malignancies and platelet disoders. 2 The Task Force also is eager to expand research about somatic mutations. Certain hematologic diseases have been very well-studied in terms of finding pathogenic mutations, Dr. Ebert said. However, this research is far from complete. “The Task Force is looking for op- portunities to support research about under-studied disease states,” Dr. Ebert said. “We also want to aggregate all of the complex somatic data that have been collected and make these easily accessible by researchers and clinicians.” REFERENCES 1. Mullighan CG. The ASH Agenda for Hematology Research: a roadmap for advancing scientific discovery and cures for hematologic diseases. Blood Advances. 2018;2:2430-2. 2. American Society of Hematology. ASH announces partnership with the University of North Carolina, a ClinGen grantee, to curate genomic data for blood disease research. April 5, 2018. Accessed November 2, 2018, from http://www.hematology.org/Newsroom/Press- Releases/2018/8460.aspx. 146 ASH Clinical News tailoring the treatment to an individ- ual. That is something hematologist/ oncologists have been doing for a long time,” said Dr. Mullighan, who also is chair of the ASH Committee on Scientific Affairs and co-chair of ASH’s Task Force on Precision Medicine. Equipped with more precise tools for understanding and treating disease, clinicians can predict which patients will be susceptible to certain disease and which patients are more likely respond to specific treatments. “Within the past decade – with greater access to large-scale tumor sequencing and the identification of potentially actionable genetic changes – the idea of precision medicine has become much more fashionable,” Dr. Mullighan added. Fashionable, yes, but maybe not accessible. The reality is that preci- sion medicine is not yet considered part of the routine care of most patients with cancer, explained Louis M. Staudt, MD, PhD, director of the Center for Cancer Genomics at the National Cancer Institute (NCI). That is because cancer is “an incredibly complex set of diseases that are essentially the result of a ‘Darwinian evolution’ of individual malignant cells within a patient that occurs typically over decades,” he said. “The challenge in bringing preci- sion medicine to every patient is to truly understand which genetic changes in cancer are important drivers of the malignant process and which are nonfunctional passenger mutations,” Dr. Staudt said. “In the future, I can envision and hope for a time when we can not only sequence a patient’s tumor but also point to evidence that shows which mutations are important to pay at- tention to and which will respond to a particular drug.” But, in most cases, the science is not there yet, Dr. Staudt said. Precision Medicine Progress In some cases, though, a focus on understanding the genomic land- scape of a disease has improved the diagnosis and treatment of certain blood cancers in recent years, Dr. Mullighan said. In the pediatric arena, clini- cians now have a better grasp of the drivers that classify subtypes of acute lymphocytic leukemia (ALL), which have helped them better select treatments. He offered the follow- ing example: About 10 percent of patients with childhood ALL have Philadelphia chromosome–like disease. About 10 percent of these cases will have ABL-class fusions that respond to ABL1 tyrosine kinase inhibitors, and another 30 percent will have mutations that activate the JAK-STAT pathway that may respond to JAK inhibitors. 4 “Precision medicine is more than matching a drug to a mutation; it is tailoring the treatment to an individual.” —CHARLES G. MULLIGHAN, MBBS (Hons.), MSc, MD Increased use of sequencing also has revealed a large number of inherited polymorphisms and muta- tions that are associated with drug response, resistance, and toxicity and the risk of leukemic transformation. He noted, though, that these discov- eries are a culmination of a decade’s worth of work, not just on research done since the Moonshot launched. “At St. Jude, we sequence all patients with this diagnosis now,” Dr. Mullighan said. “We don’t wait until they relapse.” The U.S. Food and Drug Admin- istration (FDA) also has approved targeted therapies for acute my- eloid leukemia (AML), noted Lucy Godley, MD, PhD, professor in the department of medicine, section of hematology/oncology at The Univer- sity of Chicago. For her day-to-day practice, the biggest precision medicine success story is the approval of midostaurin. The FLT3 inhibitor, in combination with chemotherapy, is indicated for the treatment of patients with newly diagnosed, FLT3-positive AML – approximately one-third of the adult AML population, generally consid- ered to be a poor-risk mutation. 5 “I now consider midostaurin standard therapy,” Dr. Godley said. She added that investigators are looking into whether midostaurin can be given more broadly, “because evidence has suggested that other leukemias could have FLT3 activated by other mechanisms.” She also applauded the approv- als of the IDH inhibitors ivosidenib and enasidenib, which are indicated for adult patients with relapsed or refractory IDH1- or IDH2-mutated AML. The FDA based its approval of ivosidenib on results from a single- arm study of 174 adults in which ap- proximately 33 percent experienced complete response or complete response with partial hematologic recovery. 6 Enasidenib was approved based on results from a single-arm study of 199 patients, in which 23 percent had complete remission or complete remission with partial hematologic recovery. 7 Both drugs are approved with a companion diagnostic. “In leukemia, it is common to have point mutations of IDH1 or IDH2 genes,” Dr. Godley explained. “There are many older people who have AML who wouldn’t be candi- dates for induction chemotherapy because of age or comorbidities who are now able to be treated with a pill. That is pretty remarkable.” Precision medicine also has enabled researchers to identify powerfully prognostic mutations in myelodysplastic syndromes (MDS), according to Benjamin L. Ebert, MD, PhD, chair of medical oncology at Dana-Farber Cancer Institute in Boston. “Given that MDS is a heterog- enous disease and patient prognosis varies widely, these mutations are useful in helping to predict progno- sis,” Dr. Ebert said. In a recent New England Journal of Medicine paper, for example, researchers found that patients with MDS who harbored at least one persistent disease-associated mutation 30 days after undergo- ing allogeneic hematopoietic cell transplantation had higher rates of progressive disease and lower rates of progression-free survival at one year, compared with patients who had mutation clearance. 8 “Knowing this information may provide an opportunity for earlier intervention to delay or prevent progression and also may [help] identify patients who can be recommended for more aggressive monitoring,” study co- author Meagan Jacoby, MD, PhD, from the Washington University School of Medicine in St. Louis, told ASH Clinical News. 9 Missing in Actionable Precision medicine efforts have made next-generation sequencing easier, faster, and cheaper to perform – exponentially increasing the amount of information clinicians have about December 2018